Peer-reviewed publications:

Miller BH, Schultz LE, Gulati A, Pletcher MT. Phenotypic characterization of a genetically diverse panel of mice for behavioral despair and anxiety. PLos One 2010.

Miller BH, Schultz LE, Gulati A, et al. Haplotype -based genetic genetic analysis of behavioral despair in inbred mouse strains. BMC Biology. 2009

Schneyer AL, Sidis, Y, Gulati, A et al. Differential Antagonism of Activin, Myostatin, and GDF11 by Wild Type and Mutant Follistatin. Endocrinology 2008.

Miller, B.H., Schultz, L.E., Gulati, A, et al. Genetic Regulation of Behavioral and Neuronal Responses to Fluoxetine. Neuropsychopharmacology 2008.

Miller, B.H., Gulati, A,, and Pletcher, M.T. Strain-dependent behavioral and neuronal responses to fluoxetine. Neuroscience. Abstr., 2006

Oral/Poster Presentations:

Fleisch, S, Gulati, A, Watson,A. UNC Homeless Support Program: Developing a Street Outreach Community Rotation for Psychiatry Residents. International Street Medicine Symposium. October 2013. Boston, MA

Schneyer, A, Sidis, Y., Keutmann, H., Gulati, A, Sun, J., and Krasney, P. Follistatin Binding To Activin And Myostatin Involves Partially Distinct Contact Regions: Implications For Development Of Ligand-Specific Antagonists. The Endocrine Society’s 89th Annual Meeting, Oral/Poster presentation, 2006

Thesis Research:

Gulati, A. Identification of Follistatin Mutations that Differentially Bind and Inhibit TGF-B Family Members Myostatin and Activain A (Funded by Pfizer, Inc.) Department of Reproductive Endocrinology, Harvard Medical School, 2005-2008

Thesis Research Summary: My investigations identified molecules that can inhibit myostatin, a negative regulator of muscle growth, thereby increasing muscle mass, and therefore quality of health, in patients with disease related cachexia (ex. Cancer, AIDS patients) without the side effects resulting from concurrent inhibition of related proteins.